DNA Damage Control SmartSunscreens offer an entirely new level of protection against damaging UVA and UVB rays and significantly improve the appearance of photoaged skin. Our sunscreens offer the best defense against skin photodamage, providing complete daily photoprotection.
Findings from a recent study reinforce the belief that effective photoprotection must provide more than UV coverage. Newest is the inclusion of Primary Protective Factors that absorb or reflect UV radiation and Secondary Protective Factors [antioxidants in combination with DNA repair enzymes] that disrupt the photochemical cascade, triggered by UV-penetration, to limit skin damage.
“Secondary photoprotection involves the use of active agents to interfere with or counteract the inherent photochemical processes that can induce DNA damage in skin cells. Secondary photoprotection may be achieved by an extremely heterogeneous and constantly growing group of molecules that are termed “actives”. These actives include antioxidants and DNA repair enzymes (e.g., photolyase, T4N5 and endonuclease).”
– P. Schroeder, PhD and J. Krutmann, MD
Institut für Umweltmedizinische Forschung (IUF) at the Heinrich-Heine-Universität Düsseldorf, Düsseldorf, Germany
DNA Damage Control sunscreens defend against UV insults with the newest, most powerful technology: liposome-encapsulated DNA repair enzymes. Clinical studies document the role of these enzymes in reducing the visible consequences of UV-induced damage.
- Photolysomes. A hyper-efficient liposome- encapsulated DNA repair enzyme, photolyase, derived from plankton, is unique in its ability to reduce visible signs of photodamage to skin.
- Endosomes. A liposome-encapsulated extract from the marine microbe Micrococcus Lysate. Extremely UV resistant, the extract contains the enzyme UV-endonuclease, which improves the appearance of sun-damaged skin.
- Breakthrough DNA repair technology with light-activated enzymes helps reduce the appearance of photodamage.
- Zinc oxide and titanium dioxide offer high-performance physical UVA/UVB protection.
- Octinoxate and octisalate filter UVB rays.
Every day, even in cloud cover, ultraviolet [UV] radiation assaults exposed skin. UV-induced damage is cumulative; its effects account for nearly all visible signs of premature aging and non-melanoma skin cancers.1
- UVB radiation [burning rays] inflicts damage to the skin’s superficial epidermal layers.
- UVA radiation [aging rays] penetrates skin more deeply, damaging cells where skin cancers originate.
DNA controls the genetic program of the skin and the greatest threat to it comes from the sun. Skin DNA absorbs both UVA and UVB radiation. Each incidence of exposure triggers a complex response to the damage, whether or not sunburn forms. After decades of repeated damage and response cycles, the effects surface and become visibly apparent.
The consequences of unrepaired DNA damage to the skin are enormous. In the early stages, damage appears as texture and tone loss, wrinkle formation and hyperpigmentation; in the end stages, skin cancers may result.
1. Skin Cancer Foundation, www.skincancer.org, Accessed 4/19/11
The 2015 Nobel Prize for Chemistry was awarded to three pioneering scientists who have shown how several DNA repair systems function to repair damaged DNA. The same DNA repair science that won the 2015 Nobel Prize in Chemistry is used in NEOVA groundbreaking formulas. Now that’s SmartScience, with the most advanced technology available.